The role of Nutrition in Treating Hyperactivity

Hyperactivity whitethorn vary as to source, and relationship to other conditions. These sources and relationships may be both physical and psychiatric in consultation.

Hyperactivity may be physiologic, and, thus, not associated with some(prenominal) other pathology (Chess, 1960). It may besides derive from organic school principal damage (Chess, 1960). In this context, the hyperactive child typically exhibits such(prenominal) symptoms as educational deficits, short attention span, perceptual difficulties, protective tendencies, and sleep disturbances (Chess, 1960). Hyperactivity may alike be associated with mental mental retardation where no brain damage is evident (Chess, 1960).

Hyperactivity may also be a symptom of reaction or psychoneurotic behavior disorder (Chess, 1960). In this context, the hyperactive child comm simply exhibits a somewhat devious motivational character, as partially of an attempt to cope with environmental stress (Chess, 1960). Hyperactivity may also be symptomatic of childhood schizophrenia (Chess, 1960).

Therapies (Generally) for Hyperactivity

The appropriate therapy for the hyperactive child depends to a great extent on the character of the hyperkinesis. If hyperactivity is a way of expressing anxiety stemming from an intrapsychic conflict related to aggression, psychotherapy may be the indicated treatment approach (Freedman, Kaplan, & Sadock, 1982). In this context, mental counseling for the entire family of the hyperkinet

The regimen used in the study focused "on a broad range of substances" (Kaplan, McNicol, Conte, & Moghadam, 1989, p. 8). For all subjects, food dyes, food flavors, preservatives, monosodium glutamate, chocolate, and caffein were eliminated," and "the amount of simple sugars was decreased" in the experimental fast (Kaplan, McNicol, Conte, & Moghadam, 1989, p. 8). Further, several additional substances were eliminated from the experimental diet for selected subjects, where parents indicated that such foods " skill be a problem" (Kaplan, McNicol, Conte, & Moghadam, 1989, p. 8).

Nutrition for Hyperactivity Treatment, Early Findings

Chess, S. (1960). Hyperactivity in children. New York State Journal of Medicine, 60, 23792385.

Phenylalanine: Not helpful in hyperactivity. (1987). American Journal of Nursing, 87, 1326, 1327.

Hinsie, L. E., & Campbell, R. J. (1980). Psychiatric dictionary, 5th edition. New York: Oxford University Press.

Medications may include stimulants, such as amphetamines, methylphenidate hydrochloride, and 2dimethylaminoethanol (Safer, & Krager, 1988). such stimulants, paradoxically, act as sedatives on hyperkinetic children (Morgan, 1988).

The topics indicated no statistically prodigious treatment order effects were present (Kaplan, McNicol, Conte, & Moghadam, 1989). It was also set in motion that 41.7 portion of the subjects experienced an approximate 50 percent feeler in behavior as a result of consuming only the experimental diet (Kaplan, McNicol, Conte, & Moghadam, 1989). A further 16.7 percent of subjects experienced an approximate 12 percent improvement in behavior as a result of consuming only the experimental diet (Kaplan, McNicol, Conte, & Moghadam, 1989). While the remaining 41.6 percent of the subjects experienced no behavior improvement as a result of consuming only the experimental diet, neither did they experience both regression in behavior as a mo of the experience (Kaplan, McNicol
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